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KLK11 promotes the activation of mTOR and protein synthesis to facilitate cardiac hypertrophy.
Wang Y, Liao H, Wang Y, Zhou J, Wang F, Xie Y, Zhao K, Gao W. Wang Y, et al. BMC Cardiovasc Disord. 2021 May 31;21(1):266. doi: 10.1186/s12872-021-02053-y. BMC Cardiovasc Disord. 2021. PMID: 34059001 Free PMC article.
Our in vitro experiments demonstrated that KLK11 overexpression promoted whereas KLK11 knockdown repressed cardiomyocytes hypertrophy induced by angiotensin II, as evidenced by cardiomyocyte size and the expression of hypertrophy-related fetal genes. ...Repression o …
Our in vitro experiments demonstrated that KLK11 overexpression promoted whereas KLK11 knockdown repressed cardiomyocytes hype …
Activation profiles of human kallikrein-related peptidases by proteases of the thrombostasis axis.
Yoon H, Blaber SI, Evans DM, Trim J, Juliano MA, Scarisbrick IA, Blaber M. Yoon H, et al. Protein Sci. 2008 Nov;17(11):1998-2007. doi: 10.1110/ps.036715.108. Epub 2008 Aug 12. Protein Sci. 2008. PMID: 18697857 Free PMC article.
Pro-KLK6 and 14 can be activated by both plasmin and uPA, with plasmin being the best activator of pro-KLK6 identified to date. Pro-KLK11 and 12 can be activated by a broad-spectrum of thrombostasis proteases, with thrombin exhibiting a high degree of selectivity for pro-K …
Pro-KLK6 and 14 can be activated by both plasmin and uPA, with plasmin being the best activator of pro-KLK6 identified to date. Pro-KLK11 …